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Research interests
Engineering and Technology
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Dive into the research topics where Benoît Schneider is active. These topic labels come from the works of this person. Together they form a unique fingerprint.
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Collaborations and top research areas from the last five years
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Deux nouveaux acteurs de la libération contrôlée des vésicules extracellulaires - Les protéases ADAM10 et ADAM17
Translated title of the contribution :ADAM10 and ADAM17 proteases: new players in the controlled release of extracellular vesicles Research output: Contribution to journal › Article › peer-review
Open Access -
Acrylamide-induced noradrenergic axon degeneration is promoted via a non-cell autonomous mechanism, involving microglial Tnfaip2/TNF-α and oxidative stress pathways
Zong, C., Sato, H., Schneider, B., Shichino, S., Ueha, S., Wu, B., Matsushima, K., Okayama, T., Ikeo, K., Urushitani, M., Ito, H., Iwama, S., Fergany, A., Ichihara, S., Ohsako, S. & Ichihara, G., 15 Sept 2025, In: Journal of Hazardous Materials. 496, 139125.Research output: Contribution to journal › Article › peer-review
Open Access -
ADAM Sheddase Activity Promotes the Detachment of Small Extracellular Vesicles From the Plasma Membrane
Bizingre, C., Arellano-Anaya, Z., Picard, F., Pietri, M., Baudry, A., Roussel, F., Bianchi, C., Alleaume-Butaux, A., Ardila-Osorio, H., Romao, M., Lavieu, G., Raposo, G. & Schneider, B., 1 Jul 2025, In: Journal of Extracellular Vesicles. 14, 7, e70114.Research output: Contribution to journal › Article › peer-review
Open Access -
Basic Science and Pathogenesis
Schneider, B., 1 Dec 2024, In: Alzheimer's & dementia : the journal of the Alzheimer's Association. 20, p. e087445Research output: Contribution to journal › Review article › peer-review
Open Access -
Post-translational modifications in prion diseases
Bizingre, C., Bianchi, C., Baudry, A., Alleaume-Butaux, A., Schneider, B. & Pietri, M., 1 Jan 2024, In: Frontiers in Molecular Neuroscience. 17, 1405415.Research output: Contribution to journal › Review article › peer-review
Open Access -
Targeting the chemokine receptor CXCR4 with histamine analog to reduce inflammation in juvenile arthritis
Bekaddour, N., Smith, N., Beitz, B., Llibre, A., Dott, T., Baudry, A., Korganow, A. S., Nisole, S., Mouy, R., Breton, S., Bader-Meunier, B., Duffy, D., Terrier, B., Schneider, B., Quartier, P., Rodero, M. P. & Herbeuval, J. P., 1 Jan 2023, In: Frontiers in Immunology. 14, 1178172.Research output: Contribution to journal › Article › peer-review
Open Access -
Déviation du métabolisme énergétique et neurodégénérescence dans les maladies à prions
Translated title of the contribution :Corruption of energy metabolism and neurodegeneration in prion diseases Research output: Contribution to journal › Article › peer-review
Open Access -
Titanium dioxide and carbon black nanoparticles disrupt neuronal homeostasis via excessive activation of cellular prion protein signaling
Ribeiro, L. W., Pietri, M., Ardila-Osorio, H., Baudry, A., Boudet-Devaud, F., Bizingre, C., Arellano-Anaya, Z. E., Haeberlé, A. M., Gadot, N., Boland, S., Devineau, S., Bailly, Y., Kellermann, O., Bencsik, A. & Schneider, B., 1 Dec 2022, In: Particle and Fibre Toxicology. 19, 1, 48.Research output: Contribution to journal › Article › peer-review
Open Access -
Loss of prion protein control of glucose metabolism promotes neurodegeneration in model of prion diseases
Arnould, H., Baudouin, V., Baudry, A., Ribeiro, L. W., Ardila-Osorio, H., Pietri, M., Caradeuc, C., Soultawi, C., Williams, D., Alvarez, M., Crozet, C., Djouadi, F., Laforge, M., Bertho, G., Kellermann, O., Launay, J. M., Schmitt-Ulms, G. & Schneider, B., 1 Oct 2021, In: PLoS Pathogens. 17, 10, e1009991.Research output: Contribution to journal › Article › peer-review
Open Access -
Stem Cells to Decipher the Physiological Roles of 5-HT2B Receptor Signaling
Baudry, A., Kellermann, O., Launay, J. M., Piétri, M. & Schneider, B., 1 Jan 2021, Receptors. Humana Press Inc., p. 53-70 18 p. (Receptors; vol. 35).Research output: Chapter in Book/Report/Conference proceeding › Chapter › peer-review
Open Access